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To deepen assess the impact of gpoA deletion on bacterial pathogenesis, we examined the intracellular survival of the gpoA mutant in three different host cell types: human pneumocytes (A549

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This suggests that this pathway is not active in neonatal mouse hearts and is dispensable for cardiomyocyte cell cycle exit 51

The most important finding in the present study was that the BHV (i.e., by brain MRI finding at days 2 and 7) and BHA (i.e., by histopathological finding at day 14) were significantly reduced in DPP4 D-HS and F344 HS + Sita animals than in F344 HS counterparts after AHS

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