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Similarly, PNPLA3 I148M promotes hepatosteatosis through ABHD5-dependent inhibition of PNPLA2 (75), suggesting that small molecules that target the PNPLA3 I148M/ABHD5 interaction could be potential therapeutics for FLD and diabetes

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The research so far suggests that GPRC6A likely plays a role in the coordination of GLP-1 release but is not essential for it

Thus, GLP-1 signaling represents a promising target for PD treatment

10.1111/obr.13210 101 van GastelJ.HendrickxJ