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An increase in IGF-1 in the circulation, in the liver and also in the muscle was observed in the Goat -/- mice, either as a result of the disordered GH pulse pattern, or because there was a failure of the elevated IGF-1 levels to feedback on GH release

10.1007/s00726-015-2125-8 5

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3,4 In parallel, renal failure also predisposes to poorer glycaemic control (both hyper- and hypoglycaemia) because of an altered renal physiology, which disrupts normal glucose and insulin metabolism