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This membrane-active mechanism provides broad-spectrum bacteriostatic coverage against Staphylococcus aureus, Escherichia coli, Pseudomonas aeruginosa, and other environmental contaminants likely to be introduced through multiple vial access events

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Moreover, serotonin metabolites (5-hydroxyindoleacetic acid) derived from activated platelets and mast cells in the lung can act as ligands to drive eosinophil recruitment via the chemotactic receptor G protein-coupled receptor 35, exacerbating fungal infections (Romero et al., 2015)