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Pro-inflammatory macrophages and effector T cells predominantly rely on glycolysis to sustain their inflammatory functions, whereas anti-inflammatory macrophages and regulatory T cells preferentially utilize oxidative phosphorylation and fatty acid oxidation ( With advances in immunometabolism research, multiple glucose metabolism-related signaling pathways have been identified as contributors to the pathogenesis of myocarditis, including hypoxia-inducible factor-1 (HIF-1) ( In recent years, therapeutic strategies targeting glucose metabolic reprogramming have demonstrated potential benefits in multiple experimental models of myocarditis (32)

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Inositol works at the cellular level and is thought to prevent the formation of cancer and may even shrink pre-existing cancers

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