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This cross-talk mechanism among signaling pathways provides a theoretical basis for understanding OS as a potential modulator of hormone signaling (Figure 3)

Although GPX4 depletion alone failed to affect HSCs function in vivo , Vav1- Gpx4 knockout mice fed with a vitamin E-depleted diet exhibited a reduced number of HSCs and dysregulated hematopoiesis, mainly due to increased ferroptosis [22]

Choline, which distributes cholesterol and prevents it from getting deposited in one part of the body

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