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Several other studies have demonstrated that butyrate can regulate gut microbiota, hepatic Glucagon-like peptide-1 (GLP-1) receptor expression, TLR4 pathways and intestinal tight junctions thus attenuating the development of MASLD (125128)

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EPCs contribute to postnatal vasculogenesis, and are recruited from the bone marrow to sites of injury via growth factors, cytokines and hypoxia-related signaling pathways, where they differentiate into mature endothelial cells and incorporate themselves into sites of active neovascularization [41, 51]