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This, in turn, protects the BBB and reduces neuroinflammatory responses (Dalile et al., 2019)

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Overall, our results confirm the involvement of well-established mediators (e.g., GNAS, RELA, PPARG) while pointing to less-characterized interactors (e.g., TSHB, GNB5) that may contribute to the pleiotropic effects of incretin-based therapies