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Such thalamic damage results in delayed neuronal recovery (154)

Originally developed for treating type 2 diabetes, it has shown an unexpected but beneficial side effect: significant weight loss

We conclude: (i) activation of G s -coupled TGR5 receptors causes stimulation of PI hydrolysis, and release of GLP-1 and PYY via a PKA-independent, cAMP-dependent mechanism involving Epac/PLC-/Ca 2+ pathway, and (ii) H 2 S has potent inhibitory effects on GLP-1 and PYY release in response to TGR5 activation, and the mechanism involves inhibition of PLC-/Ca 2+ pathway

This premise is based on the notion that subzero temperatures in supercooled storage can substantially slow down metabolism to mitigate the metabolic storage lesion and thus extend storage time

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