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Excessive apoptosis accelerates the decline in ovarian reserve, a phenomenon particularly evident in conditions such as DOR, premature ovarian insufficiency (POI), and premature ovarian failure (POF)

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In the absence of the cluster, such monothiol glutaredoxins can still be glutathionylated 26,27,29 and might (slowly) transfer the glutathione moiety to reduced cysteine residues as suggested for the AtGrxS15-dependent oxidation of reduced roGFP2 by GSSG