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The activation of RhoA via knocking down its negative regulator was reported to be associated with decreased surface AMPARs (Shen et al., 2020), and impaired LTP was observed in the RhoA-GAP, oligophrenin-1 (OPHN1) KO mice (Kasri et al., 2009)
Because NO can suppress IDO expression and promote its degradation [166], the NO-scavenging activity of 3-HAA may also indirectly sustain KP flux, highlighting the complex feedback loops linking inflammation, redox balance, and TRP metabolism in MDD
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Veterinary experience may provide opportunities for gathering additional safety and efficacy data that could potentially inform human applications
By influencing tyrosinase signaling, it helps regulate melanin synthesis before excess pigment becomes fixed within the epidermis
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