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The Breast and Prostate Cohort Consortium (BPC3), a collaboration of large US and European Cohorts, genotyped a total of 1416 SNPs for 24 genes involved in the IGF-1 pathway in 6,292 Caucasian postmenopausal women with diagnosed BrCa as compared to 8,135 controls and did not find any SNP associations with BrCa risk [39]

GSH and GSSG levels were measured as previously described (21)

According to some studies (Table 1), SeMet could inhibit cell proliferation by inducing ROS generation and activating apoptosis cellular proteins, including the caspase family and p53 (73, 74)

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