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As D23 could potentially form hydrogen bond with the activator, the loss of activity observed in the mutant supported such a model

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UV radiation can cause cell damage by generating ROS and inflammatory mediators, which directly attack the DNA of skin cells (102)

Despite improvements in small molecule antioxidant levels, a putative upregulation in the PCMT1 system might suggest proteome-specific oxidant stress in E-Sol5 and at supercooled temperatures that is alleviated by methionine-fueled pathways

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