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A single nucleotide polymorphism in sh2b3/lnk promotes hypertension development and renal damage

It has been suggested that the brain energy deficit-mitochondrial-oxidative stress axis may represent a key pathway in migraine pathogenesis (8)

To replicate the situation where efficacy predictions are made before treatment, we assumed the dose, treatment duration and drug type variables were unknown, and imputed these values in the model to an arbitrary constant value for all participants