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Previously, our lab has constructed transposon mutant libraries in K56-2, a multidrug-resistant ET12 epidemic lineage clinical isolate 26 , to identify the essential genome 27 and characterise targets and mechanisms of action for antimicrobials 28,29,30

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Potential regulation of energy utilization

this phenomenon has been linked with the increased resistance at doxorubicin treatment due to the lack of CT2 in the cell membrane that, in turn, impairs drug entry into cancer cells (Novak et al., 2015)

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