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Smith, J.A., et al

2008 doi: 10.1073/pnas.0800612105

Similarly, TGR5 activation elevates intracellular cAMP levels, leading to downstream activation of protein kinase A (PKA) and exchange protein directly activated by cAMP (Epac), which suppresses NF-B signaling, inhibits NLRP3 inflammasome activation, and can promote macrophage polarization from the pro-inflammatory M1 phenotype toward the anti-inflammatory M2 phenotype 148

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Its influence extends beyond individual neurons to the gut microbiota, with studies showing that SFN modulates gut bacteria linked to ASD symptoms