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In contrast, induction of ER stress via central administration of thapsigargin completely abolished the RYGB-mediated improvement in leptin sensitivity, without affecting control animals, highlighting ER stress as a key regulatory factor in the beneficial effects of RYGB on hypothalamic leptin signaling [97]

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Preiss D, Tikkanen M, Welsh P, Ford I, Lovato L, Elam M, LaRosa J, DeMicco DA, Colhoun H, Goldenberg I, Murphy M, MacDonald T, Pedersen T, Keech A, Ridker P, Kjekshus J, Sattar N, McMurray J