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Intraperitoneally administered ethanol also resulted in higher plasma concentrations in the presence of GLP-1R agonism, demonstrating that the ethanol-metabolizing enzymes present in the intestinal epithelium are not major contributors to this GLP-1R agonism effect on blood ethanol levels (Fig

19 One report compared semaglutide with other GLP-1 RAs (dulaglutide, exenatide) and with non-GLP-1 RA antihyperglycemics (SGLT-2i, DPP4i, sulfonylurea)

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