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Surprisingly, mice with specific genetic deletion of Gpx1 appear phenotypically normal and with normal life span (Ho et al., 1997), suggesting that there are alternative compensatory mechanisms for H 2 O 2 scavenging in Gpx1 deficiency
However, they suggest that ALCAR may be an important antidepressant therapy, either alone or in conjunction with other mood enhancers, especially in those with other medical conditions who may not tolerate high dose antidepressants
doi: 10.3390/antiox10010064 24 NewsholmePCruzatVFKeaneKNCarlessiRde BittencourtPIH
This difference may reflect sample size variations rather than true dose response relationships
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