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Conclusion Taken together, ample evidence from animal and human studies in hypertension shows a clear association with increased vascular oxidative stress, attributable to the activation of several ROS sources including Nox, xanthine oxidase, mitochondrial respiratory chain and uncoupled eNOS

THE ANTI-CELL DEATH ROLE OF GSH The anti-cell death functions of GSH are mediated through coordinated control of oxidative stress, redox-sensitive signaling, and cysteine-based protein modification, thereby setting the threshold for commitment to regulated cell death