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Interdisciplinary approaches that involve mitochondrial signaling in resilience, aging, and metabolism are needed, and perhaps mitochondria should be defined as social organelles (Picard and Sandi, 2021)

We now demonstrate that GIPR agonist administration critically relies on Gipr expression in the hindbrain, while GIPR antagonism relies on hypothalamic Gipr expressionboth are probably neuronal populations, given that we used hSyn-driven Cre-expression for the region-specific knockout studies