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In the glutaminolysis-fueled cytoplasmic NADPH-generating pathway (Figure 2E), there are 4 major mechanisms available to restore the mitochondrial matrix NADPH oxidized by IDH2

Therefore, this highlights the need for further establishing and validating the crosstalk between NAD + and the different features of mitochondrial function as a panel of possible molecular efficacy readouts in clinical settings

The lag between biochemical stimulation and observable density is why early discontinuation is the most common protocol failure

Characterization of the GSTU10-10 isoenzyme revealed that it has an antioxidant catalytic function by acting as a hydroperoxidase and has a very low K m for GSH suggesting that GSTU10-10 is able to perform efficient catalysis under conditions where GSH concentrations are low, e.g., during oxidative stress (Skopelitou et al., 2015)