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SLE, characterized by chronic inflammation, induces endothelial cell damage and dysfunction ( Figure 1 Macrophage SRs (CD36, SR-A1, LOX-1) mediate oxLDL uptake, forming foam cells essential for AS progression ( The buildup of lipids, immune cells, and debris along with extracellular matrix (ECM) degradation and rising inflammation promotes unstable plaque development ( 2.2 Type I IFN response Type I interferons (IFN-I) are crucial in the development of SLE-Related AS, as depicted in Figure 2
Helps improve workout endurance and stamina
Groups at risk of riboflavin inadequacy include vegan athletes and pregnant and breastfeeding women and their babies
13 C-NMR studies by Scafidi et al
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