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NTR1 agonists can exert neuroprotective effects and improve cognitive function by alleviating excessive microglial activation and inhibiting oxidative stress, cell apoptosis, and synaptic loss in the hippocampus [67, 68]

Thats where GLP-1 medications can help

Behavioral endpoints such as food intake patterns and satiety-proxy measures are also common

The median fasting GLP-1 in the low versus high GLP-1 groups in the prediabetes and T2D subjects were 16.06 pmol/L versus 27.28 pmol/L and 21.37 pmol/L versus 30.39 pmol/L respectively