Its largely due to the treatments (antiretroviral therapies), which can lead to a preferential accumulation of visceral fat through a mix of mechanisms, including causing mitochondrial dysfunction and insulin resistance
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As a class, however, these agents work similarly where they mimic the structure of GLP-1 in vivo , and thus, can bind the GLP-1R, but are not subject to degradation by DPP-4
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And the lower inflammation associated with better glutathione levels helps break the inflammatory cycle that often accompanies metabolic issues
