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Patients with muscular dystrophies and other myopathies are at additional risk because membrane instability and rhabdomyolysis release large quantities of intracellular potassium and myoglobin

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These include the expression and characterization of metalloenzyme paralogs and cambialistic metalloenzymes recently described (Section 4) and specific mechanistic features, recently uncovered, of metal delivery to downstream protein clients and trafficking of metals across bacterial membranes, including intracellular organelles (see Section 5)

Supplement formats continue to evolve, and delivery methods have become just as important as the ingredients themselves