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Mitochondrial reactive oxygen species (ROS) act as a key regulator, promoting RIPK1 autophosphorylation, which in turn enhances necrosome assembly and MLKL pore-forming capacity, ultimately driving necroptosis (32)

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In contrast, induction of c-Myc mRNAs occurred earlier, and their levels remained higher at several time points in the Gstp1/2 / regenerating livers compared with the WT control livers (Figure 5a)