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In addition, increasing plasma levels of Apo C-III, which inhibits the activity of LPL in individuals with diabetes, can reduce the catabolism of VLDL.[] Additionally, increasing the transfer of fatty acids to the liver, a common disorder in insulin-resistant diabetes, may increase the secretion of VLDL,[ , ] and also its concentration may rise by higher amounts of fatty acids and Apo B100.[] By stimulating and contributing to the decomposition of fatty acids in the mitochondria, L-carnitine can probably reduce the number of fatty acids to produce Lp (a) in patients who are likely to have high atherogenic lipoprotein production

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