Once deubiquitinase is suppressed, caspase-dependent apoptosis and GPX4-degradation-dependent ferroptosis is activated, contributing to the accumulation of ubiquitination proteins that facilitates cell death (Yang L
Through its interaction with actin, Thymosin Beta 4 facilitates cellular movements and orchestrates the repair of damaged tissues, making it a promising therapeutic agent for various conditions
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pylori included the pathways related to autophagy and ferroptosis, providing a new direction for the exploration of H