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Insufficient or disorganized TJ complex formation, an expression of claudin-1 with decreased levels of claudin-5 trans interaction, and claudin-5/ZO-1 interaction in leaky vessels (219) are among the proposed mechanisms behind an incomplete recovery of BBB permeability

MOTS-c modulates the FOXO transcription factors and regulates cellular senescence, the state in which aged or damaged cells stop dividing but refuse to die, instead secreting a cocktail of inflammatory signals known as the senescence-associated secretory phenotype (SASP)