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107 had baseline FibroScan data, with mean kPa of 7.2 and CAP of 322
(PubMed) Smith AD, Smith SM, de Jager CA, et al
As research in the field of ASD continues, it is becoming clear that the etiology of most ASD cases involves complicated interactions between genetic predisposition and environmental exposures or triggers
3319-A HOST PRECONDITIONING AND TRANSIENT MITOGEN EXPRESSION VIA mRNA-LNP LEAD TO ROBUST PRIMARY HUMAN HEPATOCYTE ENGRAFTMENT AND TRANSIENT iPSC-DERIVED HEPATOCYTE SURVIVAL IN A MURINE MODEL OF AATD LIVER DISEASE Anna R Smith 1 , Fatima Rizvi 1 , Elissa Everton 1 , Anisah Adeagbo 1 , Hua Liu 1 , Ying Tam 2 , Norbert Pardi 3 , Drew Weissman 3 and Valerie Gouon-Evans 1 , (1)Boston University School of Medicine, (2)Acuitas Therapeutics, (3)University of Pennsylvania Perelman School of Medicine Background: Alpha-1 antitrypsin deficiency (AATD) is a genetic disease caused by a single base pair mutation of the SERPINA1 gene and increases risk of liver and lung disease
Serum bile acid levels and gene expression were modulated in a circadian-dependent manner, consistent with Linafexor's short half-life and metabolic rhythms
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