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Results VRK2-deficient PC cells are vulnerable to GSH inhibition To investigate the impact of VRK2 on metabolic reprogramming, we treated VRK2-wild-type (VRK2-WT) and VRK2-knockout (VRK2-KO) CFPAC-1 cell lines with 281 metabolism compounds (Supplementary Table1)

C., Smith, A

Correlation between SNP and Hcy in study subjects Experimental evidence showed that GPx-1 regulates Hcy-induced cardiovascular risk and that Hcy attenuates the ability of the cell to detoxify hydrogen peroxide by inhibiting the intracellular activity of GPx-1 [29]

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