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The far-reaching scope of neuroinflammation after traumatic brain injury

Prenatal exposure to maternal infection alters cytokine expression in the placenta, amniotic fluid, and fetal brain

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These mechanistic perspectives provide potential explanations for some of the clinical improvements seen and illustrate the various pathways through which FMT might exert its effects on disease processes

Overall, our findings suggest that MG binds to GSH, inhibits GSH resynthesis, and affects crucial enzyme activities that are involved in GSH synthesis and GSH-related selenoprotein generation, thereby promoting ferroptosis (Figure 5I)