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Free Radical Biology and Medicine, 19 (2), 227250

targeted EC cells with miR-302a overexpressing EXOs, which inhibited their division and migration by lowering cyclin D1 levels and deactivating the AKT signaling (Li et al., 2019)

UCMSCs-EXOs combine the benefits of hUCMSCs pluripotency with nanoscale dimensions, improving their therapeutic potential through longer circulation half-life

Strom and colleagues published a retrospective cohort study using an electronic database of medical records from outpatient medicine practitioners in the United Kingdom to assess risk factors for sulfonamide cross-reactivity [3]

10.1007/s43032-025-01889-5 240 MenonU.Gentry-MaharajA.BurnellM.SinghN.RyanA.KarpinskyjC.et al (2021)